A year ago in a scientific source, Cell, a study was forwarded to the public that offered empirical epistemological evidence that a hormone called betatrophin, of Angiopoietin-like protein-8, could ramp up pancreatic B-cell proliferation in a mouse model of insulin resistance. The results made quite a splash, the studys authors that was led by Doug Melton at Harvard University even wrote that betatropin treatment could argument or replace insulin injections by increasing the number of endogenous insulin-producing cells in diabetics. A follow-up study by an independent group of researchers found that the hormone isnt required for B-cell function or growth. Viktoria Gusarova of Regeneron Pharmaceuticals and her colleagues wrote in the Cell paperwork hypothesis linked in October 23, added this, The lack of expansion of the beta cell area could theoretically be due to simultaneous increases in replication and apoptosis frequencies. However, even if this were the case, it would not change our observation that Angpt 18 overexpression did not increase beta cell area, contrary to what was reported by Y., et al.
Posted on: Thu, 30 Oct 2014 21:22:06 +0000
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