Rapid Recovery Hyperbarics hbot4u 909.477.4545 Chronic Fatigue Syndrome, FM, M.S. and Lyme Disease (Mycoplasma associated disorders) The etiology of Chronic Fatigue Syndrome (CFS) and Fibromyalgia (FM) is still not entirely clear, and has been variously suggested as an infection in its own right, or as the sequel to other infections or neuro-toxic insults--notably Epstein Barr Virus (EBV) and allergic reaction to a number of substances including, but not limited to, silicone, chemical crop sprays, and petroleum products. In any event, the mechanism in physiology which causes the symptoms of chronic fatigue is insufficiently understood but can be explained by the following hypothesis or postulate which is put forward on the basis that it explains both the mechanism and the therapy. Muscular and other cellular activity requires energy, and this in turn requires glycolysis, which relies upon the burning of blood carried sugars with blood carried oxygen. The release of energy by this chemical process produces waste chemicals, notably lactates, and these in turn impose the symptoms of fatigue until they are fixed and removed by the same blood chemistry. This is the Krebs cycle normal to all life. Under ordinary circumstances, oxygen is transported to the body cells by this chemical cycle in which it combines with hemoglobin in the erythrocytes of the blood stream, and the same chemical (HGB) removes and fixes the waste lactates for disposal as carbon dioxide into the pulmonary system. In order to flow through the venous system, erythrocytes (RBCs) must be deformed, since they are larger in diameter than the veins in which they must travel. This is to ensure that a maximum surface contact area is available. In cases of Chronic Fatigue Syndrome, either the internal pressure in a percentage of the RBCs is elevated, or the permeability of their cell walls is lowered, or both. The result is that these RBCs are not able to deform and travel in the microcirculatory system. This deprives the cells, which the RBCs should serve of oxygen, and allows accumulated lactates to produce the symptoms of Chronic Fatigue Syndrome. CONCLUSIONS: The hypothesis can be supported in that if you exhaust a fit subject, his or her RBCs will behave as above roughly in the same percentages as the subject’s proportionate fatigue, but will recover to normal as the subject recovers. The Chronic Fatigue patient’s blood, on the other hand, will remain abnormal in this aspect, and the patient will remain fatigued. TREATMENT: Hyperbaric Oxygen Therapy at 2 ATA - 2.4 ATA produces an increased elasticity in the RBCs and at the same time seems to reduce the Delta P between their contents and the surrounding medium. There is also a probability that the dissolved oxygen in the plasma may oxidize whatever substance is responsible for the decrease in RBC cell wall permeability. Certainly, the most immediate effect of Hyperbaric Oxygen Therapy is to relieve the cellular hypoxia, which is a feature of Chronic Fatigue Syndrome. Sixty minutes of treatment twice daily for 30 -40 treatments, followed by weekly treatments prn seems to resolve CFS symptoms in the majority of patients, and eventually to resolve them completely. hbot4u 909.477.4545 #oxygen #hyperbaricoxygentreatment #hbot #MS #chronicfatiguesyndrome #hypoxia #FM #lymedisease
Posted on: Mon, 26 Jan 2015 02:10:50 +0000