United States Army Medical Research Institute of Infectious - TopicsExpress



          

United States Army Medical Research Institute of Infectious Diseases: Ebola is Airborne - Same Transmission as the Flu Virus Evidence now suggests the NBC cameraman contracted Ebola via airborne exposure as did the Spanish nurse. In this clip Dr. Rima Laibow CONFIRMS that Ebola is not only airborne but it is weaponized. The United States Army Medical Research Institute of Infectious Diseases (USAMRIID; pronounced: you-SAM-rid) is the U.S Army’s main institution and facility for defensive research into countermeasures against biological warfare. It is located on Fort Detrick, Maryland and is a subordinate lab of the U.S. Army Medical Research and Materiel Command (USAMRMC), headquartered on the same installation. USAMRIID is the only U.S. Department of Defense (DoD) laboratory equipped to study highly hazardous viruses at Biosafety Level 4 within positive pressure personnel suits. ---- ncbi.nlm.nih.gov/pubmed/8551825 According to the Center for Aerobiological Sciences, U.S. Army Medical Research Institute of Infectious Diseases at Fort Detrick, Maryland: (1) Ebola has an aerosol stability that is comparable to Influenza-A (2) Much like Flu, Airborne Ebola transmissions need Winter type conditions to maximize Aerosol infection Filoviruses, which are classified as Category A Bioterrorism Agents by the Centers for Disease Control and Prevention (Atlanta, GA), have stability in aerosol form comparable to other lipid containing viruses such as influenza A virus, a low infectious dose by the aerosol route (less than 10 PFU) in NHPs, and case fatality rates as high as ~90% . The mode of acquisition of viral infection in index cases is usually unknown. Secondary transmission of filovirus infection is typically thought to occur by direct contact with infected persons or infected blood or tissues. There is no strong evidence of secondary transmission by the aerosol route in African filovirus outbreaks. However, aerosol transmission is thought to be possible and may occur in conditions of lower temperature and humidity which may not have been factors in outbreaks in warmer climates [13]. At the very least, the potential exists for aerosol transmission, given that virus is detected in bodily secretions, the pulmonary alveolar interstitial cells, and within lung spaces Analysis: Its clear that when Ebola is in the air it is at least as hardy as Influenza. Its also clear that coughing and sneezing is what makes Influenza airborne; the same should be expected of Ebola. Moreover, just as sun, heat, and humidity along the Earths Equatorial regions serve to burn Influenza out of the air, the same should be expected of Ebola. The difference with Ebola is that physical contact with even the tiniest amounts of infected bodily fluid can cause infection, hence unlike flu it also readily spreads in equatorial regions. When Ebola spreads to the regions of the Earth which experience Fall and Winter Flu seasons, airborne Ebola infectious routes are to be expected in conjunction with direct contact infection. Ebola has the capability to infect pretty much every cell in the entire human respiratory tract. Similarly, our skin offers little resistance to even the smallest amounts of Ebola. How much airborne transmission will occur will be a function of how well Ebola induces coughing and sneezing in its victims in cold weather climates. Coughing and nasal bleeding are both reported symptoms in Africa, so the worst should be expected. In that regard, co-infections with Flu, Cold, or even seasonal Allergies will readily transform Ebola victims into biowarefare factories. Unlike Flu, a person need not inhale airborne Ebola to be infected via airborne transmission. Merely walking through an airspace (or touching the objects therein) where an Ebola victim has coughed or sneezed is potentially enough for a cold weather infection to occur. As such, all indicators are that Ebolas potential rate of infectious spread in cold weather climates is EXPLOSIVELY greater than what is occurring in Equatorial Africa Mutation: Given that the experts are keenly aware that most mutations lead to viral dead ends and given the ARMYs public research documents make such a clear case that the Ebola airborne risk is here and now, the question remains: why are the experts pushing a future mutation fear on the public? The primary benefits of the media mutation gambit are: 1) When the public becomes aware Ebola is airborne, the public will default to blaming a mutation rather blaming the experts for having prior knowledge of Ebolas transmissability 2) A scary future fear makes for great immediate fund raising from a public seeking to avoid it. 3) The expert clique comes down hard on experts that do anything which is perceived to immediately raise public fear, an accurate warning to the public can immediately negatively affect a forthright experts budget and prestige 4) Public knowledge of imminent Public Health threats negatively affects supply chains and the logistics planned responses ---- Dr. Brantly acquired Ebola while strictly following CDC guidelines (simple mask, goggles, or face shield, gloves, gown, leg covering, shoe covers), so it is likely he became infected by inhaling contaminated droplet nuclei into his lungs or having them settle into his conjunctival (eye) sacs despite the use of CDC level protection against direct contact. Dr. Brantly apparently did not use a full-face respirator with P-100 filters, but rather a simple or N-95 face mask. An interviewer noted that “Brantly says he isnt sure how he got infected. Hes certain he didnt violate any [CDC] safety guidelines.” The United States Army Medical Research Institute of Infectious Diseases conducted a monkey to monkey Ebola study in December 1995, published in The Lancet, Vol. 346. (Here is a link to the abstract, but the entire article must be purchased.) Several Rhesus monkeys were infected with Zaire Ebola by intramuscular injection while three control Rhesus monkeys were kept in cages separated 10 feet from the infected monkeys. All of the injected monkeys died of Ebola by day 13 and 2 out of 3 control monkeys died of Ebola by 8 days after that. The authors of this study concluded that: The exact mode of transmission to the control monkeys cannot be absolutely determined, although the pattern of pulmonary antigen staining in one of the control monkeys was virtually identical to that reported in experimental Ebola virus aerosol infection in rhesus monkeys, suggesting airborne transmission of the disease via infectious droplets... Fomite or contact droplet transmission of the virus between cages was considered unlikely. Standard procedures in our BL4 containment laboratories have always been successful in the prevention of transmission of Ebola or Marburg virus to uninflected animals. Thus, pulmonary, nasopharyngeal, oral, or conjunctival exposure to airborne droplets of the virus had to be considered as the most likely mode of infection... Our present findings emphasize the advisability of at-risk personnel employing precautions to safeguard against ocular, oral, and nasopharyngeal exposure to the virus. youtube/watch?v=3zb41sCOqPw
Posted on: Thu, 09 Oct 2014 03:06:11 +0000

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